The precise utilization of ABG, VBG, and pulse oximetry remains controversial. Unfortunately, there is little high-level evidence investigating whether these interventions affect patient outcomes (for example, there is precious little evidence to support most of the target values that we're chasing after). This chapter attempts to explore the strengths, weaknesses, and indications for various techniques.
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The IBCC chapter is located 👉 here.
- The podcast & comments are below.
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Great Episode! Thanks for your teaching! Greetings from Germany
Perfect timing, I was just confronted with the conflict regarding ABG vs VBG on Meddit and was looking for some good clarification. IBCC is one of my favorite references (recently helped me stabilize an afib RVR when synchronized cardioversion failed!) Major fan, thank you very much for creating these posts and the podcast.
Very useful information. I’d like to translate it to catalan language, I think it will be very useful to the catalan ER and critical care community. Is it possible?
Not completely. Depends o the amount of deadspace. That can be increased due to low cardiac output in addition to lung problems. It’s the interplay that is important. That’s why etCO2 is useful in arrest management to determine adequacy of compressions or ROSC.
It would be great if CME was available for Josh Farkas’ material. I enjoy his posts/podcasts because I work in the ICU.
Excellent information and the material on the Perfusion Index is highly applicable to my anesthetic practice.
“Septic shock may decrease the stroke volume and vascular tone – which could have unpredictable effects on the perfusion index.”
Septic shock decreases central vascular tone, but as I understand the pathophysiology, peripheral vascular tone will likely increase as compensatory mechanisms shunt blood away from the periphery.
The PI should decrease with a decrease in stroke volume and an increase in peripheral vascular tone. Or am I missing something?